Description
Title: The Verifiable Contribution of Curcumin to Oxidative Stress-Mediated Mitochondrial Dysfunction in Mammals
Abstract: The pathogenesis of a number of human diseases is linked to oxidative stress and mitochondrial dysfunction. DNA mutations in mitochondria are brought on by an excessive production of reactive oxygen species (ROS) and/or by inadequate antioxidant defenses, which harm the mitochondrial matrix. respiratory chain, and modifies the permeability of membranes and the protective mechanisms of mitochondria. Numerous diseases can develop as a result of all these changes. Numerous biological activities are displayed by curcumin, the active component of turmeric plant rhizomes (i.e., antioxidant, anti-inflammatory, anticancer, and antimicrobial). Numerous studies have demonstrated in recent years that curcumin can lessen the symptoms of oxidative stress and mitochondrial dysfunction. diseases. We go over curcumin’s antioxidant mechanism, importance in reducing oxidative stress, and suppression of mitochondrial dysfunction in mammals in this review. We also go over the gaps in the literature and offer suggestions for how curcumin research in mammals should develop going forward.
Keywords: curcumin; mammals; mitochondrial dysfunction; oxidative stress; antioxidant; anti-inflammatory; anticancer; antimicrobial
Paper Quality: SCOPUS / Web of Science Level Research Paper
Subject: Biomolecules
Writer Experience: 20+ Years
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