Description
Title: The Function of MALAT1 in the Neuroprotection of Brain from Hypoxic/Ischemic Injury
Abstract: Worldwide, hypoxic and ischemic brain injury has emerged as a serious public health issue that can lead to neurological disability and mortality. Numerous diseases are regulated in part by long-chain non-coding RNAs. The nervous system contains large amounts of the long non-coding RNA (lncRNA) known as long intergenic non-coding RNA (lincRNA), or metastatic-related lung adenocarcinoma transcript 1 (MALAT1). The fact that MALAT1 is more abundant in the brain suggests that it might be linked to crucial roles in pathophysiological processes. As a result, over the past few years, more and more information about MALAT1’s role in neuronal cell hypoxic/ischemic injury has come to light. The neuroprotective molecular mechanism of MALAT1 and its regulation of the pathophysiological processes of brain hypoxic/ischemic injury are discussed in this article in light of recent research. MALAT1 may act as a regulator by interacting with proteins or RNAs to carry out its function, making it a potential therapeutic target in cerebral ischemia/hypoxia.
Keywords: MALAT1; hypoxic/ischemic injury; neuronal cell; protection
Paper Quality: SCOPUS / Web of Science Level Research Paper
Subject: Biomolecules
Writer Experience: 20+ Years
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