Research Paper on Idiopathic pulmonary fibrosis (IPF) and allergic pneumonitis (AP) are etiopathologies involving signaling pathways and their miRNA regulators (HP)

Description

Title: Idiopathic pulmonary fibrosis (IPF) and allergic pneumonitis (AP) are etiopathologies involving signaling pathways and their miRNA regulators (HP)

Abstract: Idiopathic pulmonary fibrosis (IPF) and hypersensitivity pneumonitis (HP) are both interstitial lung diseases that are heterogeneous in nature (ILD). This group of diseases presents with complicated clinical non-specific features, making diagnosis and treatment difficult. In this review we focus on several crucial signaling pathways participating in inflammation, fibrosis and EMT processes, so important in the course of ILD: TNF-a/NFkb, TGF-b/SMAD, Wnt-b-catenin and PI3K-Akt signaling. Additionally, this review summarizes the function of specific signaling pathways and a few miRNAs that act as their regulators during the onset and advancement of IPF and HP. Recent research suggests that miRNAs may play a role as molecular markers differentiating the clinical course of ILD.

Keywords: words: idiopathic pulmonary fibrosis, hypersensitivity pneumonitis, molecular markers, pathogenesis, signaling pathways

Paper Quality: SCOPUS / Web of Science Level Research Paper

Subject: Advances in Respiratory Medicine

Writer Experience: 20+ Years

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