Description
Title: Epigenetic Effect on B-Cell Lymphomagenesis Associated with EBV
Abstract: Epigenetic changes that result in transcriptional activation or repression are crucial for the growth of human cancers. An epidemiological study found that tumor viruses are linked to about 20% of all human cancers. The first human tumor virus, Epstein-Barr virus (EBV), exhibits frequent epigenetic changes on the viral and host genomes in associated cancers, both of lymphoid and epithelial origin. Similar to how viral oncoproteins recruit epigenetic regulators in order to deregulate the cellular gene expression profile, which results in a number of human cancers, the cell type-dependent different EBV latent gene expression patterns appear to be determined by the cellular epigenetic machinery. This review explains the epigenetic effects of interactions between the EBV and host during the development of several EBV-induced B-cell lymphomas, which may help researchers develop new therapeutic approaches to treat EBV-associated B-cell lymphomas by changing reversible patho-epigenetic markings.
Keywords: Epstein-Barr virus; B-cell lymphoma; Epigenetics
Paper Quality: SCOPUS / Web of Science Level Research Paper
Subject: Biomolecules
Writer Experience: 20+ Years
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