Description
Title: Conclusions about Alcohol and Prostate Cancer Must Be Drawn
Abstract: The question of whether drinking alcohol increases the risk of developing prostate cancer has been hotly contested in scientific and medical communities for a long time (PCa). Numerous thorough studies from various nations and geographical regions have demonstrated a positive correlation between PCa development and moderate and heavy drinking. However, some findings do not support the existence of such a correlation; some even imply that drinking wine may prevent or slow the growth of prostate tumors. Here, we have thoroughly examined the evidence supporting and refuting the contribution of alcohol to the development of PCa. We discovered that many epidemiological studies neglected to take into account additional, possibly crucial factors, such as diet (particularly, low intake of fish, vegetables, and linoleic acid, and excessive use of red meat), smoking, family history of PCa, low physical activity, history of high sexual activity, especially with early age of first intercourse, and STDs. Additionally, there are differences between observations due to selectivity standards for control groups, questions about the type and amount of alcohol consumed, and inaccurate reporting of alcohol use. Given that prostate tumors typically grow slowly, the lifetime history of alcohol use is crucial. Nevertheless, many epidemiological studies that found no association only looked at recent or current drinking patterns. However, the overall finding is that excessive alcohol consumption, particularly binge drinking, is linked to an increased risk for PCa, and this effect is not specific to any one type of drink. Alcohol consumption is also directly related to PCa lethality because it may hasten prostate tumor growth and hasten the development of metastatic PCa. As a result, we advise PCa patients to stop drinking immediately. We go over the characteristics of ethanol metabolism in prostate tissue as well as how ethanol metabolites negatively affect intracellular organization and trafficking. We also discuss how drinking alcohol affects the level of prostate-specific antigen and the risk of benign prostatic hyperplasia. Finally, we discuss the mechanisms by which alcohol can interfere with prostate carcinogenesis as well as any negative effects it might have on a patient undergoing androgen deprivation therapy.
Keywords: alcohol consumption; ethanol metabolism; prostate cancer; prostate cancer-associated mortality
Paper Quality: SCOPUS / Web of Science Level Research Paper
Subject: Biomolecules
Writer Experience: 20+ Years
Plagiarism Report: Turnitin Plagiarism Report will be less than 10%
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